Perspective on the Osteoclast
Identifieur interne : 00AA72 ( Main/Exploration ); précédent : 00AA71; suivant : 00AA73Perspective on the Osteoclast
Auteurs : Frank C. Cackowski [États-Unis] ; G. David Roodman [États-Unis]Source :
- Annals of the New York Academy of Sciences [ 0077-8923 ] ; 2007-11.
Descripteurs français
- Wicri :
- topic : Mineur (ouvrier).
English descriptors
- KwdEn :
- Angiogenesis, Angiogenic, Angiogenic activity, Angiogenic factors, Antiangiogenic, Antiangiogenic effects, Bfgf, Biol, Bisphosphonates, Bone cells, Bone development, Bone matrix, Bone metastases, Bone miner, Bone resorption, Cackowski, Cackowski roodman, Cell biol, Cell types, Chem, Chick chorioallantoic membrane, Clin, Cortical bone, Cytokine, Endochondral ossification, Endothelial, Endothelial cells, Fracture, Fracture healing, Gene expression, Growth factor, Growth factors, Hypoxia, Macrophage, Matrix, Miner, Myeloma, Myeloma cells, Neutralizing antibodies, Ocls, Osteoblast, Osteoclast, Osteoclastogenesis, Osteopontin, Receptor activator, Resorption, Roodman, Trabecular bone, Vegf, York academy.
- Teeft :
- Angiogenesis, Angiogenic, Angiogenic activity, Angiogenic factors, Antiangiogenic, Antiangiogenic effects, Bfgf, Biol, Bisphosphonates, Bone cells, Bone development, Bone matrix, Bone metastases, Bone miner, Bone resorption, Cackowski, Cackowski roodman, Cell biol, Cell types, Chem, Chick chorioallantoic membrane, Clin, Cortical bone, Cytokine, Endochondral ossification, Endothelial, Endothelial cells, Fracture, Fracture healing, Gene expression, Growth factor, Growth factors, Hypoxia, Macrophage, Matrix, Miner, Myeloma, Myeloma cells, Neutralizing antibodies, Ocls, Osteoblast, Osteoclast, Osteoclastogenesis, Osteopontin, Receptor activator, Resorption, Roodman, Trabecular bone, Vegf, York academy.
Abstract
Abstract: There have been reports recently that the osteoclast stimulates angiogenesis in vitro. We review the evidence suggesting that the osteoclast directly stimulates angiogenesis, and discuss the feasibility of the hypothesis that the osteoclast can be a proangiogenic cell. Reasons supporting the feasibility of this hypothesis include proangiogenic factors produced by osteoclasts, the angiogenic effects of macrophages, the antiangiogenic effects of nitrogen containing bisphosphonates, and the physical proximity of osteoclasts to endothelial cells.
Url:
DOI: 10.1196/annals.1402.073
Affiliations:
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Le document en format XML
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<profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Angiogenesis</term>
<term>Angiogenic</term>
<term>Angiogenic activity</term>
<term>Angiogenic factors</term>
<term>Antiangiogenic</term>
<term>Antiangiogenic effects</term>
<term>Bfgf</term>
<term>Biol</term>
<term>Bisphosphonates</term>
<term>Bone cells</term>
<term>Bone development</term>
<term>Bone matrix</term>
<term>Bone metastases</term>
<term>Bone miner</term>
<term>Bone resorption</term>
<term>Cackowski</term>
<term>Cackowski roodman</term>
<term>Cell biol</term>
<term>Cell types</term>
<term>Chem</term>
<term>Chick chorioallantoic membrane</term>
<term>Clin</term>
<term>Cortical bone</term>
<term>Cytokine</term>
<term>Endochondral ossification</term>
<term>Endothelial</term>
<term>Endothelial cells</term>
<term>Fracture</term>
<term>Fracture healing</term>
<term>Gene expression</term>
<term>Growth factor</term>
<term>Growth factors</term>
<term>Hypoxia</term>
<term>Macrophage</term>
<term>Matrix</term>
<term>Miner</term>
<term>Myeloma</term>
<term>Myeloma cells</term>
<term>Neutralizing antibodies</term>
<term>Ocls</term>
<term>Osteoblast</term>
<term>Osteoclast</term>
<term>Osteoclastogenesis</term>
<term>Osteopontin</term>
<term>Receptor activator</term>
<term>Resorption</term>
<term>Roodman</term>
<term>Trabecular bone</term>
<term>Vegf</term>
<term>York academy</term>
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<term>Angiogenic</term>
<term>Angiogenic activity</term>
<term>Angiogenic factors</term>
<term>Antiangiogenic</term>
<term>Antiangiogenic effects</term>
<term>Bfgf</term>
<term>Biol</term>
<term>Bisphosphonates</term>
<term>Bone cells</term>
<term>Bone development</term>
<term>Bone matrix</term>
<term>Bone metastases</term>
<term>Bone miner</term>
<term>Bone resorption</term>
<term>Cackowski</term>
<term>Cackowski roodman</term>
<term>Cell biol</term>
<term>Cell types</term>
<term>Chem</term>
<term>Chick chorioallantoic membrane</term>
<term>Clin</term>
<term>Cortical bone</term>
<term>Cytokine</term>
<term>Endochondral ossification</term>
<term>Endothelial</term>
<term>Endothelial cells</term>
<term>Fracture</term>
<term>Fracture healing</term>
<term>Gene expression</term>
<term>Growth factor</term>
<term>Growth factors</term>
<term>Hypoxia</term>
<term>Macrophage</term>
<term>Matrix</term>
<term>Miner</term>
<term>Myeloma</term>
<term>Myeloma cells</term>
<term>Neutralizing antibodies</term>
<term>Ocls</term>
<term>Osteoblast</term>
<term>Osteoclast</term>
<term>Osteoclastogenesis</term>
<term>Osteopontin</term>
<term>Receptor activator</term>
<term>Resorption</term>
<term>Roodman</term>
<term>Trabecular bone</term>
<term>Vegf</term>
<term>York academy</term>
</keywords>
<keywords scheme="Wicri" type="topic" xml:lang="fr"><term>Mineur (ouvrier)</term>
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<front><div type="abstract">Abstract: There have been reports recently that the osteoclast stimulates angiogenesis in vitro. We review the evidence suggesting that the osteoclast directly stimulates angiogenesis, and discuss the feasibility of the hypothesis that the osteoclast can be a proangiogenic cell. Reasons supporting the feasibility of this hypothesis include proangiogenic factors produced by osteoclasts, the angiogenic effects of macrophages, the antiangiogenic effects of nitrogen containing bisphosphonates, and the physical proximity of osteoclasts to endothelial cells.</div>
</front>
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<region><li>Pennsylvanie</li>
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<name sortKey="Roodman, G David" sort="Roodman, G David" uniqKey="Roodman G" first="G. David" last="Roodman">G. David Roodman</name>
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